Department of Animal Pathology & Clinical Pathology

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About Department of Animal Pathology & Clinical Pathology

Facts about Department of Animal Pathology & Clinical Pathology

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15

Publications

6

Academic Staff

Programs

Master - Pathology

It is through studying courses with a total of 24 study units to study compulsory and elective courses over a period of 2 to 3 semesters, followed by specialized scientific research with a total of 6 study units for the thesis. The legal period required to obtain a master’s degree ranges from 24 months...

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Master - Laboratory diagnosis

It is through studying courses with a total of 24 study units to study compulsory and elective courses over a period of 2 to 3 semesters, followed by specialized scientific research with a total of 6 study units for the thesis. The legal period required to obtain a master’s degree ranges from 24 months...

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Who works at the Department of Animal Pathology & Clinical Pathology

Department of Animal Pathology & Clinical Pathology has more than 6 academic staff members

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Ms. samira ahmed saleh shlayek

samira shlayek is one of the staff members at the department of 2 faculty of 6. He is working as a since 2015-12-30. He teaches several subjects in his major and has several puplications in the field of his interest.

Publications

Some of publications in Department of Animal Pathology & Clinical Pathology

Chondroblastic Osteosarcoma in a Cat: Case Report

Osteosarcoma is one of the most important bone tumors of human beings and pet animals. It is a rapidly progressive, early metastasize, osteolytic, highly fatal tumor. It may arise from central medullary osteoblasts, peripheral periosteal or perichonderal cells, and extra skeletal tissues or may have a pluripotent potential. A Shirazi Native breed cat was examined for the presence of multilocular lobulated partially encapsulated masses in the external ear. The excised tissue revealed anaplastic, chondroblast and osteoblast mesenchymal differentiations associated with hypercellularity, pleomorphism and increase in mitosis, consistent with chondroblastic osteosarcoma.
Seham ALAzreg(1-2016)
Publisher's website

Cellular and molecular etiology of hepatocyte injury in a murine model of environmentally induced liver abnormality

Exposures to a wide variety of environmental substances are negatively associated with many biological cell systems both in humans and rodents. Trichloroethane (TCE), a ubiquitous environmental toxicant, is used in large quantities as a dissolvent, metal degreaser, chemical intermediate, and component of consumer products. This increases the likelihood of human exposure to these compounds through dermal, inhalation and oral routes. The present in vivo study was aimed to investigate the possible cellular and molecular etiology of liver abnormality induced by early exposure to TCE using a murine model. The results showed a significant increase in liver weight. Histopathological examination revealed a TCE-induced hepatotoxicity which appeared as heavily congested central vein and blood sinusoids as well as leukocytic infiltration. Mitotic figures and apoptotic changes such as chromatin condensation and nuclear fragments were also identified. Cell death analysis demonstrates hepatocellular apoptosis was evident in the treated mice compared to control. TCE was also found to induce oxidative stress as indicated by an increase in the levels of lipid peroxidation, an oxidative stress marker. There was also a significant decrease in the DNA content of the hepatocytes of the treated groups compared to control. Agarose gel electrophoresis also provided further biochemical evidence of apoptosis by showing internucleosomal DNA fragmentation in the liver cells, indicating oxidative stress as the cause of DNA damage. These results suggest the need for a complete risk assessment of any new chemical prior to its arrival into the consumer market. Keywords: Apoptosis, DNA damage, Environmental toxicant, Liver, Oxidative stres
Seham ALAzreg(9-2016)
Publisher's website

Endocardiosis and congestive heart failure in a captive ostrich (Struthio camelus)

A seven-year-old blue-necked male ostrich was found dead after a few days of illness. The animal was living in an open yard of 25 square meters along with three other females. They were given concentrate-rich ration with free access to green leaves and water. Autopsy revealed cardiac enlargement due to left ventricular hypertrophy and right ventricular dilatation. The left aterioventricular valves were irregularly thickened and contracted. The lungs were engorged with blood and the liver had nutmeg appearance. The small intestine showed segmental sub-serosal petechial hemorrhages. Histological examination revealed myxomatous degeneration of the left aterioventricular valves, pulmonary congestion and edema, congestion of periacinar hepatic zone and fatty degeneration of outer zones, renal glomerulosclerosis and arteriosclerosis. The affected parts of the small intestine showed villous atrophy with lacteal distention. The venules in the affected intestinal segment were severely dilated while the arterioles had narrow lumen and irregular wall thickening with hyaline deposition. The current article reports an endocardiosis in ostrich and discusses other vascular disorders. Keywords: Congestive heart failure, Endocardiosis, Myomatous valvular degeneration, Ostrich.
Seham ALAzreg(11-2013)
Publisher's website

Journals

Journals published by Department of Animal Pathology & Clinical Pathology