Faculty of Vet Medicine

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About Faculty of Vet Medicine

The Faculty of Veterinary Medicine was established in 1975. It was the first Faculty of Veterinary Medicine in Libya. It is one of the citadels of science and knowledge at the University of Tripoli. This scientific institution works around the clock to meet the needs of the community of veterinarians and contributes to supporting the national economy. It values the care for animal health. It maintains increasing animal production, preserving human health and protecting the environment.

Facts about Faculty of Vet Medicine

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167

Publications

85

Academic Staff

245

Students

23

Graduates

Faculty of Vet Medicine Announcements

2022-05-17
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Who works at the Faculty of Vet Medicine

Faculty of Vet Medicine has more than 85 academic staff members

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Dr. HUDA HASSAN AL MUKTAR AL GRIW

هدى القريو هي احد اعضاء هيئة التدريس بقسم الاحياء الدقيقة والطفليات بكلية الطب البيطري. تعمل السيدة هدى القريو بجامعة طرابلس كـمحاضر منذ 20-06-2012 ولها العديد من المنشورات العلمية في مجال تخصصها

Publications

Some of publications in Faculty of Vet Medicine

Spreading of Salmonella enteritidis in the cecum of chickens

Adhesion and colonization of high (2 × 108 CFU) and low doses (2 × 102 CFU) ofSalmonella enteritidis (phage type 4) was determined in the ceca collected 6 h-4 weeks after inoculation (pi), of 1-d-old White Plymouth Rock orally-inoculated chickens.S. enteritidis was associated with the epithelial surface of the villi in the low-dose group 18 h-7 d pi, the penetration in the cecal lamina propria was observed on day 1 and 10 pi. In the high-dose group, adhesion and colonization was observed in all birds killed 6 h-14 d pi; penetration of the bacteria into the cecal lamina propria was seen 1–21 d pi. Large numbers of macrophage-like cells containingS. enteritidis were observed in the cecal lamina propria on days 3–21 pi. Colonization and migration byS. enteritidis in the intestinal tract of chickens was shown to be dose dependent.
A. A. Asheg(3-2003)
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Molecular detection and characterisation of avian paramyxovirus type 1 in backyard chickens and pigeons in Alzintan city of Libya

Abstract Avian paramyxovirus-1 (APMV-1) is the causative agent of Newcastle Disease which affects many species of birds leading to high mortality and heavy economic losses among poultry industry worldwide. Newcastle disease is endemic in Libya with frequent outbreaks occurring in commercial and backyard poultry. APMV-1 was isolated and characterised during the outbreak in 2013. In current study, we report another Newcastle disease outbreak that emerged in backyard chickens and pigeons in Alzintan city on March 2015. Two viruses were detected in cloacal swabs from backyard chickens, namely APMV-1/Libya/15VIR5368/2015 and APMV-1/Libya/15VIR5371/2015. Genetic sequencing of these viruses revealed the presence of velogenic APMV-1 belonging to genotype VIIi genetically similar to the viruses isolated on 2013. During the same period, neurologic signs and mortality were noticed in pigeons. Samples of brain tissue were tested by rRT-PCR which revealed presence of velogenic APMV-1 belonging to lineage 4A (GKKRKR*F Lin.4A) or genotype VIb. To the best of our knowledge, this is the first report of the detection and molecular characterization of APMV-1 in a pigeon in Libya. The phylogenetic analysis of the F gene showed 86% identity to isolates from Iran and Egypt. This study may indicate the circulation of APMV-1 within backyard birds and pigeons which may present a threat to commercial poultry. Considering these findings, vaccination of backyard birds and pigeons and further epidemiological studies are strongly strongly recommended. arabic 19 English 111
Abdulwahab Kammon, Isabella Monne, Abdulatif Asheg , Giovanni Cattoli (1-2018)
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Système IGF et croissance fœtale.

La croissance fœtale est un processus complexe dépendant de facteurs génétiques, environnementaux, nutritionnels et hormonaux d’origine maternelle, placentaire et fœtale. Le système IGF est l’un des systèmes hormonaux les plus importants pour la régulation de la croissance fœtale et placentaire [1]. Le gène IGF-II est régulé par le phénomène d’empreinte parentale et est exprimé seulement à partir de l’allèle paternel dans la majorité des tissus pendant la vie fœtale. Les gènes soumis à empreinte parentale sont régulés de manière spécifique et sont particulièrement vulnérables aux signaux environnementaux et nutritionnels. La dérégulation d’un groupe de gènes de la région 11p15 soumise à empreinte parentale, incluant le gène IGF-II, est responsable de deux pathologies de croissance fœtale (les syndromes de Silver-Russell, OMIM 180860 et de Wiedemann-Beckwith, OMIM 130650) qui ont une présentation phénotypique opposée. Ces deux syndromes représentent d’excellents modèles de pathologies humaines pour l’étude de la régulation de l’empreinte parentale. arabic 9 English 26
- Demars, J , S. Rossignol, Mansur Ennuri Moftah Shmela, I. Netchine, S. Azzi, A. El-Osta, Y. Le Bouc, C. Gicquel(1-2012)
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