كلية الطب البيطري

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حول كلية الطب البيطري

تأسست كلية الطب البيطري عام 1975م كأول كلية للطب البيطري في ليبيا. تعد الكلية من قلاع العلم و المعرفة الهامة بجامعة طرابلس ومؤسسة علمية تلبي إحتياجات المجتمع من الأطباء البيطريين وتساهم في دعم الإقتصاد الوطني من خلال العناية بصحة الحيوان وزيادة الإنتاج الحيواني والمحافظة على صحة الإنسان وحماية البيئة.

حقائق حول كلية الطب البيطري

نفتخر بما نقدمه للمجتمع والعالم

224

المنشورات العلمية

87

هيئة التدريس

191

الطلبة

1684

الخريجون

المزيد من الأخبار

البرامج الدراسية

ماجستير في الأحياء الدقيقة
تخصص الأحياء الدقيقة

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التفاصيل
ماجستير في أمراض الطفيليات
تخصص علم الطفيليات

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التفاصيل
ماجستير في تخصص الجراحة والتخدير والأشعة
تخصص الجراحة والتخدير والأشعة

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التفاصيل

من يعمل بـكلية الطب البيطري

يوجد بـكلية الطب البيطري أكثر من 87 عضو هيئة تدريس

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أ.د. عبدالناصر صالح رجب ديهوم

عبدالناصر ديهوم هو احد اعضاء هيئة التدريس بقسم الطب الوقائي بكلية الطب البيطري. يعمل السيد عبدالناصر ديهوم بجامعة طرابلس كـأستاذ مشارك منذ 2015-06-01 وله العديد من المنشورات العلمية في مجال تخصصه

منشورات مختارة

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Fertility and Reproductive Outcome in Mice Following Trichloroethane (TCE) Exposure

Exposure to trichloroethane (TCE), an industrial solvent, has been shown to be negatively associated with reproductive performance. The present study was performed to assess the effects of TCE exposure on the reproductive performance and outcome in mice during a critical developmental window of later reproductive life. A group of female mice were injected intraperitoneally twice weekly for three weeks with TCE (100 and 400 μg/kg). Mice were followed up for signs of toxicity and death. Changes in uterine tissues have also been investigated by histopathology. The results showed that TCE exposure has reduced the number of F0 fertile females comparing to controls. Moreover, TCE exposure resulted in a decreased pups number and changed sex ratio in the litter of F0 TCEtreated dams. Histopathological examination revealed a TCEinduced uterine toxicity appeared as a severe endometrial hyperplasia with squamous cell metaplasia and adenomyosis. These results indicate that TCE exposure during a critical reproductive developmental window could affect the fertility and interfere with the reproductive outcome in mice. Keyword: TCE; fertility; reproductive outcome; uterus; mice.
Seham ALAzreg(1-2016)
Publisher's website

Green tea modulates nicotine toxicity on kidney in Wistar albino rats

The present study investigated the effects of an aqueous extract of green tea (Camellia sinensis) on kidney functions through Urea, uric acid and creatinine measurement within male Wistar albino rats intoxicated with nicotine. Forty male Wistar albino rats were randomized into four groups of ten. Over a 28 day period the control group received daily subcutaneous dose of saline (10 ml) and drank only water, the green tea group received subcutaneous dose of saline (10 ml) and drank only green tea, the nicotine group received subcutaneous dose of nicotine (10 ml saline with 3mg/kg weight of nicotine) and drank only water, and the green tea with nicotine received subcutaneous dose of nicotine (10ml saline with 3mg/kg weight of nicotine) and drank only green tea. Urea, uric acid and creatinine were measured by ultraviolet spectrophotometer utilizing standard methods. No differences were observed between the control and green tea only groups, and no differences were found for creatinine levels between any of the groups. The Nicotine group had urea and uric acid concentrations significantly (P < 0.05) greater than those of the control, green tea only groups, and the green tea with nicotine groups. The results of the present study indicate that an aqueous extract of green tea reduced the effects of nicotine on urea and uric acid concentrations. arabic 10 English 59
Marwan Mustafa Ali Draid(6-2016)
Publisher's website

HDAC2/3 inhibitor MI192 mitigates oligodendrocyte loss and reduces microglial activation upon injury: A potential role of epigenetics

Background: During development, oligodendrocyte (OL) lineage cells are susceptible to injury, leading to life-long clinical neurodevelopmental decits, which lack effective treatments. Drugs targeting epigenetic modications that inhibit histone deacetylases (HDACs) protect from many clinical neurodegenerative disorders. Aim: This study aimed to investigate the therapeutic potential of histone deacetylase 2/3 (HDAC2/3) inhibitor MI192 on white matter (WM) pathology in a model of neonatal rat brain injury.Methods: Wistar rats (8.5-day-old, n = 32) were used to generate brain tissues. The tissues were cultured and then randomly divided into four groups and treated as following: group I (sham); the tissues were cultured under normoxia, group II (vehicle); DMSO only, group III (injury, INJ); the tissues were exposed to 20 minutes oxygen-glucose deprivation (OGD) insult, and group IV (INJ + MI192); the tissues were subjected to the OGD insult and then treated with the MI192 inhibitor. On culture day 10, the tissues were xed for biochemical and histological examinations. Results: The results showed that inhibition of HDAC2/3 activity alleviated WM pathology. Specically, MI192 treatment signicantly reduced cell death, minimized apoptosis, and mitigates the loss of the MBP+ OLs and their precursors (NG2+ OPCs). Additionally, MI192 decreased the density of reactive microglia (OX−42+). These ndings demonstrate that the inhibition of HDAC2/3 activity post-insult alleviates WM pathology through mechanism(s) including preserving OL lineage cells and suppressing microglial activation. Conclusion: The ndings of this study suggest that HDAC2/3 inhibition is a rational strategy to preserve WM or reverse its pathology upon newborn brain injury. Keywords: Brain injury, Epigenetics, MI192, Microglia, Oligodendrocyte
Mansur Ennuri Shmela, Mohamed A. Al-Griw, Emad M. Bennour(8-2021)
Publisher's website

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